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As with any hormone-modulating therapy, treatment decisions should be individualized and based on a careful assessment of potential risks, expected benefits, medical history, and treatment goals
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Several mechanisms have been suggested [5,17,33,34]: (1) reduced intrinsic factor secretion, (2) decreased vitamin B12 release from protein complexes, (3) microbial overgrowth due to slowed intestinal transit, and (4) decreased vitamin B12-intrinsic factor complex receptors
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