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It does this by scavenging reactive oxygen species (ROS), which helps protect mitochondrial components such as proteins, lipids, and DNA from oxidative damage, thereby preserving mitochondrial function
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Targeting RB1 may disrupt the control of the cell cycle in MDSCs, leading to reduced proliferation and accumulation within the TME, thereby weakening their capacity to inhibit anti-tumor immune responses by lymphoid and myeloid cells [31]
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