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Increasing glutathione levels by a novel posttranslational mechanism inhibits neuronal hyperexcitability - ScienceDirect

The emerging role of ferroptosis in the pathophysiology of neuroprogressive diseases

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In addition, given that the factors that induce ferroptosis in these different autoimmune diseases remain largely unclear, cytokines such as TNF-α and IFN-α regulate ferroptosis in different manners and contribute in various ways to disease pathogenesis (70, 81)