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Mechanistically, LPS directly activated NLRP3 inflammasome through the TLR4-TRIF-MyD88-RIPK1-FADD-CASP8 axis without eliciting pyroptotic cell death ( FIGURE 1 4 NLRP3 inflammasome in the pathogenesis of AP AP is an inflammatory disorder characterized by excessive activation of pancreatic enzymes due to diverse etiological factors, leading to autodigestion, edema, hemorrhage, and necrosis of pancreatic parenchyma and adjacent tissues