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The CtAtm1 inw-opn-occl conformation also triggered us to revisit the initial cryo-EM data collected for wild-type CtAtm1 in the presence of ATP and without supplementation of cargo
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Based on these findings, we hypothesize that the ability of Erastin to bypass the ferroptosis resistance of EMS tissue and effectively induce ferroptosis and lesion atrophy may be attributed to its unique ferroptosis-inducing mechanism and its multifaceted regulation of iron homeostasis
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