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Interestingly, ligand activation of LXRs not only activates transcription of target genes, but also inhibits transcription from promoters of certain genes of proinflammatory cytokines that do not contain LXREs, a phenomenon referred to as trans-repression ( The anticipated anti-neuroinflammatory and neuroprotective effects of B12, as predicted through transcriptome analysis in infected animals treated with the vitamin, were successfully confirmed at the histological level ( Figures 4 and 1 , respectively)

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