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and as a microbicidal peroxidase, it can induce the strong response of oxidation at the site of inflammation by catalyzing hypochlorous acid ( NF-κB regulates many genes in the inflammatory response that can be promoted by proinflammatory stimuli, such as CS and TNF-α ( in vivo experiment showed that GHK-Cu blocked the transcriptional activity of NF-κB through suppression of NF-κB by altering the phosphorylation of its inhibitor i-κBα
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