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Key lipids, including ceramides, sphingomyelins, phospholipids, diacylglycerols, and triglycerides, as well as branch-chain amino acids, have been found to be dysregulated in both ME/CFS and GWS (Germain et al., 2019

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This intermediate genotype may be associated with a moderate change in leptin receptor responsiveness, often representing a middle-ground pattern between the two homozygous genotypes