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The second part involves 20–30% of the cell death pathway and has characteristics of oxidative glutamate toxicity, because it can be inhibited specifically by vitamin E, group I metabotropic receptor agonists, a caspase inhibitor, elevated extracellular cystine, and the removal of extracellular glutamate (Schubert and Piasecki, 2001)
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doi: 10.1038/sj.leu.2404061 281 ClarkRAHuangSJMurphyGFMolletIGHijnenDMuthukuruMet al
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