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The use of MK-801, an NMDAR antagonist, was shown to inhibit the increase in Ca 2+ influx in T cells, showing the role of this receptor in the control of Ca 2+ influx in lymphocytes
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The exact mechanism by which EDTA-induced lead chelation delays renal impairment is not known, but it is hypothesized that chronic low-level lead exposure may increase ROS levels, which inactivate nitric oxide (NO)
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