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The activity of this pathway in the liver is markedly impaired or absent in the fetus and newborn infant, in cirrhotic patients, and in patients with homocystinemia (Mato et al., 1994)

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No Biossomos, o GHK‑Cu faz parte de um complexo biossomal integrado, sendo incorporado a vesículas sintéticas biomiméticas que imitam a comunicação celular