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In summary, VNS engages vagovagal and intrinsic GI neuroimmune reflexes that reinforce the intestinal barrier, suppress excessive inflammation, and foster a balanced microbiome, which are key elements of GI physiological homeostasis
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A similar mechanism has been described for the prion protein PrP, wherein its mislocalization to the cytosol enables aberrant binding to cytosolic ubiquitin ligase, sequestering the enzyme and reducing its functional availability—thereby contributing to neurodegeneration 82
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