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B-cell glycosyltransferase expression is known to respond to the cytokine micro-environment ( H 17 cells signal newly differentiated B cells with IL-22 and IL-23 to regulate glycosyltransferase expression, resulting in a pro-inflammatory agalactosylated, non-fucosylated IgG N-glycan ( Lyme disease has been demonstrated to destroy germinal centers of lymph nodes during early infection ( independent manner promote increased sialyation on the Fc portion of IgG leading to an immunotolerant, less pro-inflammatory IgG N-glycan repertoire ( Conclusion Using the GlycoTyper MALDI-FT-ICR imaging approach, we detected an unexpected IgG N-glycan signature in humans during LD
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