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Low-grade, transient oxidative signals generated during ozone therapy — including lipid electrophiles such as 4-hydroxynonenal — modify Kelch-like ECH-associated protein 1 (Keap1) and promote nuclear factor erythroid 2-related factor 2 (Nrf2) nuclear translocation, which in turn supports the GSH system at four coordinated levels: cystine import via the cystine/glutamate antiporter (xCT), de novo synthesis via glutamate–cysteine ligase, regeneration of reduced glutathione by glutathione reductase, and supply of nicotinamide adenine dinucleotide phosphate (NADPH) from the pentose phosphate pathway
pdp-attribute::No Artificial Flavors
Strengths:10 MG
cerevisiae and identified target genes to improve ERG yield
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